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Ravasz, D
It is possible to explain the role of mediators using an experimental model of autoimmune encephalitis: Protection against the experimental disease occurs after administration of a glutamate antagonist [10]
Particularly, the relevance of reactive dopamine metabolite 3,4-dihydroxyphenylacetaldehyde (DOPAL) in catechol-induced neurotoxicity
SIRT6 optimizes the metabolic state of glucose and lipids by controlling glycolysis and gluconeogenesis, affecting insulin secretion and action, and regulating lipid catabolism, transport, and synthesis
