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Conclusion This article provides a comprehensive review of the pivotal roles of immunometabolism and oxidative stress in cancer and aging, as well as their interplay
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stimulated human placental, visceral fat, and subcutaneous fat tissue explants with tumor necrosis factor (TNF), and the study showed that punicalagin significantly inhibited the TNF-induced expression of pro-inflammatory factors (IL-1, IL-6) in human placenta and adipose tissue (Nguyen-Ngo et al., 2020)