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In the context of ARDS, ferroptosis contributes to the destruction of alveolar and endothelial cells, leading to increased vascular permeability, pulmonary edema, and impaired gas exchange
doi: 10.1371/journal.pone.0196411
Taken together, the results corroborated that GSH inhibited PSC activation via down-regulation of the TGF-/SMAD pathway
In a rat model of ISO-mediated myocardial infarction, CoQ10 preconditioning not only markedly expanded cardiac GSH levels and reduced the concentration of lipid peroxidation but also alleviated myocardial damage [177]
