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Taken together, these observations suggest that hyperhomocysteinemia favors thrombogenesis
Morgan AE, Mooney KM, Wilkinson SJ, Pickles NA, Mc Auley MT
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GSH metabolism contributes to FA tolerance in DNA-repair deficient cells Finally, we reasoned that the reaction between FA and GSH forming HSMGSH molecules and their metabolization by ADH5 might limit free FA
