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METH stimulates the release of excess glutamate from neurons while inhibiting its uptake by astrocytes, leading to the overactivation of NMDARs ( tThe excitotoxicity induced by METH also triggers neuroinflammation and amplifies its neurotoxic effects

Observation of calcium microdomains at the uropod of living morphologically polarized human neutrophils using flash lamp-based fluorescence microscopy

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This ascertains an essential process not hampered with the initial tendon healing process as similar to fracture healing [83], nor ossicles formation in other tissues as it is with the application of the bone morphogenetic proteins (BMPs) [84,85,86]