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Additionally, mitochondrial-derived peptide-c (MOTS-c) has been shown to reverse mitochondrial dysfunction and reduce oxidative stress, thereby alleviating metabolic reprogramming, decreasing beta-oxidation, and increasing glycolytic flux influenced by abnormal metabolism during myocardial infarction [464]
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Differential association between biomarkers of subclinical inflammation and painful polyneuropathy: results from the KORA F4 study
