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The mechanisms underlying doxorubicin-induced cardiotoxicity involve oxidative stress and reduced antioxidant substances in cardiac tissues
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Abnormal lipid metabolism also affects the expression of various factors (LOX-1, HIF-1, VEGF-A, NOX-1, NOX-2), activating oxidative stress and causing endothelial dysfunction (ETDF)
The average annotation efficiency was determined to be 19.42% (Table S5)
