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Description
Conclusions We hypothesize that the high-folatelow vitamin B-12 interaction represents a novel cause of vitamin B-12 depletion caused by high folic-acid intake and consequent reduction in serum holoTC (Figure 11)
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doi: 10.1016/s0891-5849(03)00391-5
We solved this bioavailability challenge by sourcing S-Acetyl L-Glutathione, where the acetyl group acts as a protective shield
