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12,13 It is thus hypothesised that, in the presence of DMF, the ability of dendritic cells to properly present auto-antigens, including the aforementioned LL37, to T-lymphocytes is blocked

The specific At CNIHs can enhance the activity of GLRs (mainly AtGLR3.3) by regulating their sorting, trafficking, location, and quantity without any ligands, which in turn contribute to cytosolic Ca 2+ homeostasis, thus regulating pollen germination and pollen tube growth (Wudick et al., 2018)

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In addition, A deposition and excessive tau phosphorylation in the brains of AD patients can further inhibit the expression of SLC7A11, exacerbating GSH depletion and GPX4 inactivation, and forming a vicious cycle of oxidative stress-ferroptosis-AD pathological damage