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Specifically, dihydroartemisinin reduced Fe 2+ levels at an early stage and triggered iron autophagy, resulting in the degradation of iron autophagy-related proteins, FTH1 and NCOA4, followed by an increase in Fe 2+ levels (YU, 2021)

Enzyme Cofactor: Supports antioxidant and detox pathways through enzymes such as glutathione peroxidases and glutathione S-transferases

Jin et al., 2017)
