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Description
Bax protein is a proapoptotic member that acts by forming pores on the mitochondrial outer membrane, allowing cytochrome c to translocate into the cytoplasm, resulting in the loss of energy production and the activation of proteolytic cascade leading to apoptosis (Westphal et al., 2014)
GDAP1 exemplifies the consequences of OMM impairment, as pathogenic variants of this protein disrupt mitochondrial dynamics, calcium buffering, redox signaling, and communication between organelles
NAD+ precursors: a questionable redundancy
Iuchi K et al
