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Description
It exists purely for gastrointestinal adaptation
This is not a claim that genetics predicts exact supplement response, but rather provides a framework for more informed discussion
They are GLUT4 translocators and PI3K generates phosphatidylinositol 3,4,5-triphosphate (PIP3) that recruits AKT and activates AKT phosphorylates leading to GLUT4 translocation (widerska et al., 2021)
In CIRS, excessive complement activation, particularly of the C4a and C3a fragments, signifies persistent innate immune stimulation triggered by biotoxin residues or unresolved infection
