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Homocysteine from disease biomarker to disease prevention
(Removed: "Therapeutic Role in Brain Disorders") Drge W, Breitkreutz R
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This mutant failed to rescue TBK1 and IRF3 phosphorylation upon TLR3 activation, indicating that the catalytic activity of ANKIB1 indeed resides in its RBR and that the polyubiquitin chains it generates are essential for TBK1 activation (Fig
