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Prenatal fetal brain anomalies have been observed, such as cerebellar hypoplasia and ventriculomegaly and ventricular asymmetry, periventricular cystic lesions, corpus callosum anomalies (thin or absent), absent septum pellucidum, basal ganglia and midbrain anomalies and cystic cavitations, mega cisterna magna, hypoplastic cerebellum, cerebellum vermian anomalies or absence, optic nerve hypoplasia at different stages of gestational age (50

doi: 10.1042/BJ20091334

These Rb-independent roles for cyclin D-Cdk4,6 in promoting cell proliferation raise the question as to what is the in vivo function of the cyclin D-Cdk4,6-Rb interaction

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