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It is designed to selectively disrupt the FOXO4-p53 interaction in senescent cells, enabling their targeted clearance while sparing healthy cells in preclinical models
A phosphorylation coevolution network independent of protein abundance was also built in this study
doi: 10.3181/00379727-100-24576
J Affect Disord 143(13):3438 Stroup DF, Berlin JA, Morton SC et al (2000) Meta-analysis of Observational Studies in EpidemiologyA Proposal for Reporting
