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Results of a previous study have revealed that miR-148a-3p acts as a tumor suppressor in CRC through targeting SLC7A11 and activating ferroptosis (85)
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A 2015 study by Huang and colleagues in Drug Design, Development and Therapy used a rat alkali-burn model (n=10 per group) and HUVECs treated with BPC-157 at 200800 ng/mL, reporting accelerated wound closure with enhanced granulation tissue, reepithelialization, and dermal remodeling by day 18, alongside increased endothelial migration and vascular tube formation in vitro via ERK1/2 phosphorylation though the abstract does not report numeric % wound closure or p-values
New Phytol, 183,95105,1469-8137 77
