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In acute inflammation, dopamine synthesis can be stimulated by the upregulation of BH4 production, while chronic inflammation leads to oxidative loss of BH4 and consequent reduction in dopamine synthesis (Fanet et al., 2021)

(2023) indicated that this interaction enhances macrophage phagocytic activity and NK cell cytotoxicity, thereby bolstering the hosts first line of defense against viral and bacterial infections (238)
