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They stated that the lower enzyme activity in the HBV-infected carcinoma tissue resulted from a further disruption in the antioxidant defense system (double insults of hepatocellular carcinoma and HBV infection)
Following subchronic cagrilintide treatment, rats, but not mice, show alterations in Calcr/Prlh -expressing cells suggesting a role for this cell population in the mechanism of weight loss (PREPRINT 49 )