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have shown that the absence of FGF-2 leads to a faster regeneration/differentiation of oligodendrocytes after cuprizone-induced demyelination (Armstrong et al., 2002)

[143] showed that Salmonella enterocolitis induced preferential damage to glutamatergic myenteric neurons expressing specific non-canonical inflammasome machinery and pointed to a neuroprotective role of ENS-associated tissue-resident macrophages mediated through an arginase 1-polyamine axis