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Description
It was demonstrated that this phenotype is essentially due to a decrease in the biosynthesis of indole glucosinolates (IG
References Rosenblum, D., Joshi, N., Tao, W., Karp, J
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In addition, the absence or decrease in levels of cytosolic GSH resulted in PDI becoming more oxidized, limiting the ability of the enzyme to isomerize nonnative disulphide bonds
