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[7] [9] As with all parenteral medications, administration should be avoided in the presence of local or systemic infection at the injection site

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The results showed that both studies met this goal, with the therapy resulting in a significantly lower annualized rate of exacerbations relative to the placebo: by 30% in BOREAS and by 34% in NOTUS

GLP-1R/GIPR/GCGR Triple-Agonists Treatment Tri-agonist analogs, which activate the glucagon, GLP-1, and GIPRs, have been developed with the aim that the additional GIP activity will further offer better protection from glucagon-induced hyperglycemia