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Description
In this model, cerebral injury correlated with an increase in oxidative stress markers and accelerated caspase 3 activation
Thus, natural killer cells isolated from ethanol-fed mice exhibited preferential migration in response to chemokines binding to the CXCR3 receptor but reduced migration in response to chemokines binding to the CCR2, CXCR4, and CX3CR1 receptors
Of note, none of these studies in humans reported any adverse effects of NAC
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