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Kountourakis P, Correa AM, Hofstetter WL, Lee JH, Bhutani MS, Rice DC , Komaki R, Maru DM, Ross WA, Vaporciyan A, Swisher SG, Ajani JA
Recently, genome-wide CRISPR/Cas9-mediated loss-of-function screens showed that loss of TP53, BAX or PMAIP1 renders AML cells resistant to Venetoclax 20
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At the same time, metabolic shift towards aerobic glycolysis, altered lipid metabolism, and glutamine dependency due to oncogenic signaling and hypoxic stress, further promotes tumor survival and therapy resistance (123)
