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paragallinarum on GR for oxidative stress mitigation and virulence maintenance not only emphasizes its vital role in bacterial survival but also suggests that impairing GR function could significantly reduce pathogenicity

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Clinical data from LS patients also highlight marked gliosis as a characteristic finding, potentially resulting from reactive processes secondary to neuronal damage but it could be also a result of an increased propensity of NPCs to differentiate into astrocytes due to LS-causative mutations and mitochondrial dysregulation