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Additionally, ADSC-EV-mediated inflammatory suppression has been reported as a major underlying mechanism for treating atopic dermatitis [249, 250], sepsis [251, 252], and atherosclerosis-induced vascular lesions [253], along with other conditions of immunosuppression failure listed in this review

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Thyroid hormone controls carnitine status through modifications of -butyrobetaine hydroxylase activity and gene expression
