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63 In these three models, the mice were infected with 23 LD 50 of influenza virus and N-acetylcysteine was administered as a single daily dose of 1,000 mg/kg
Care 23 (1), 407
CHAC1 inhibition via CRSPR/Cas9 mediated knock-in of an enzyme inactivating mutation demonstrates a novel strategy to preserve muscle glutathione levels under wasting conditions but fails to prevent muscle wasting in mice
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