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Animal model studies have preliminarily revealed that Clostridium colonization can curtail the overgrowth of pro-inflammatory microbiota, restore the intestinal mucosal barrier, and limit systemic translocation of bacterial LPS, ultimately reestablishing gut microbial homeostasis (206, 207)

Interestingly, Cdc42 is indispensable in AT2, whereby it allows for differentiation into AT1 rather than remaining in the PATS state 23

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