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And this brings us back to the most critical point of all: your source
Interestingly, a few years ago it has been published that in flies missense mutations of the SLC25A39 homologue shawn results in accumulation of ROS as well as in mitochondrial dysfunction, synaptic defects, and neurodegeneration
Epidermal growth factor receptor and integrins control force-dependent vinculin recruitment to E-cadherin junctions
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