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These vasodilatory mechanisms are diverse but share two distinct characteristics in that the activity of each of the differing mechanisms increases in direct response to increasing intensities of exercise and those mechanisms all initiate the synthesis of nitric oxide (NO)

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I2=86%) and are also a useful tool for ruling out diastolic dysfunction.7 Interestingly, lower levels of NT-proBNP are found in patients with HFpEF in comparison with those with HF with reduced ejection fraction (HFrEF),8,9 possibly because of the association of NPs with end-diastolic wall stress that appears to be lower in HFpEF.10 Obokata et al

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