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Pathological activation under cytotoxic stimuli (ischemia, inflammation, or shear stress) induces calcium overload through pore-forming domains, subsequently triggering mechanotransduction pathways that disrupt organelle homeostasis (ER-mitochondrial crosstalk and UPR regulation) [22, 39, 40]

Transport of butyryl-L-carnitine, a potential prodrug, via the carnitine transporter OCTN2 and the amino acid transporter ATB(0,+) Am J Physiol Gastrointest Liver Physiol

Further studies are required to evaluate its potential as a biomarker

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