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Description
These are transient, reversibly dormant cells with the capability to act as a reservoir for disease relapse

Thus, collectively, excessive L-Glu levels can contribute to neuronal damage and degradation through different aetiology processes, including oxidative stress, excitotoxicity, mitochondrial dysfunction, inflammation, and protein aggregation, and thereby contribute to the progression of NDDs [37,38,39,40]

A weak, not clinically relevant effect has been highlighted on cycle length and hormonal status

Mitoenergetic failure in Alzheimer disease
