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Ferroptosis and autoimmune diseases Autoimmune diseases impact approximately 89% of the global population, yet the underlying mechanisms remain inadequately explored [623]

reported in a mouse model of renal ischemiareperfusion injury that oxidative signals were markedly increased in the injured kidney, whereas this phenomenon was abolished in gp91 phox /NOX2-deficient mice, suggesting that myeloid cell-mediated, NOX2-dependent oxidative burst may contribute to the establishment of the renal oxidative microenvironment in AKI and thereby promote tissue injury progression (64)

The current conversation should be medically grounded

Grell M, Douni E, Wajant H, Lhden M, Clauss M, Maxeiner B, et al