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In fact, reports of bacterial endotoxin in the pelvic cavity and menstrual blood of people with endometriosis 37 have led to a bacterial contamination hypothesis suggesting endotoxin/bacteria in the menstrual effluent contributes to pelvic inflammation, growth, and progression of endometriotic lesions (reviewed in 38,39 )

Induction of desensitization Desensitization was induced as previously described [24]

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Ethacrynic acid also inhibits L-type voltage-dependent and store-operated calcium channel , leading to relaxation of airway smooth muscle (ASM) cells