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Recently it has been demonstrated that the inhibition of the plasma membrane Na + /Ca 2+ exchanger (NCX) either by KB-R7943 (2-[2-[4-(4-nitrobenzyloxy) phenyl]ethyl] isothiourea methanesulfonate) or by the substitution of Li + for Na + in the external physiological solution considerably enhances the Ca 2+ -dependent desensitization of NMDARs [11]

Bning Journal: Molecular therapy : the journal of the American Society of Gene Therapy Journal ranking: Q1 Key takeaways: Tropism-modified AAV vectors with novel receptors significantly improve cutaneous gene therapy by increasing cell entry efficiency and targeting v8 integrin, overcoming safety concerns.