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Concurrent ROS and Ca 2+ dysregulation promote excessive MCU-dependent mitochondrial Ca 2+ uptake, lowering the threshold for mPTP opening and amplifying phosphatidylserine (PS) exposure and procoagulant activity (Melchinger et al., 2019
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Activation of soluble guanylate cyclase by carbon monoxide and inhibition by superoxide anion
However, it has been previously shown that the cause for decreased GSH formation in type 2 diabetes is not reduced synthesis of GSH, but the inability to reduce GSSG to GSH [11]
