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In conclusion, the in vivo work on Gstp / mice fully demonstrates that GSTP acts as a negative regulator in sepsis pathophysiology, and recombinant GSTP protein treatment to mice may significantly improve survival in mouse CLP sepsis model

A -catenin/TCF-coordinated chromatin loop at MYC integrates 5 and 3 Wnt-responsive enhancers

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By enabling more predictive and mechanistic investigations, they hold significant promise for advancing the preclinical development of Epitalon as a multifunctional therapeutic candidate for the management of diabetes and its systemic complications