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The involvement of IL-17A and Th17 cells in the preservation of endometrial cell viability is achieved through the suppression of apoptosis and the inhibition of NK cell-induced cytotoxicity toward endometrial cells, mediated by the ERK1/2 pathway (82)
Biochim Biophys Acta 1559:171178 Richman PG, Meister A (1975) Regulation of gamma-glutamycysteine synthetase b nonallosteric feedback inhibition by glutathione
Khanolkar, M
