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In conclusion, future research should leverage single-cell multi-omics to decipher repair heterogeneity among immune cell subsets and construct dynamic DNA damage maps to reveal the spatiotemporal relationship between lesion accumulation and inflammatory signaling
52,53 The complex mechanisms controlling the process of neutrophil phagocytosis are controversial
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To this end, recent work by Jin and colleagues explored the use of a Tat - syn-degron peptide to bind alpha-synuclein and induce its proteasomal degradation specifically
