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This is because TNSALP accounts for a significant percentage of total ALP activity, particularly in the extracellular environment
To confirm whether GPX4 is involved in the mechanism by which Se protects Cd-induced ferroptosis, we generated AML12 stable cell lines in which GPX4 expression was knocked down (Figure 5E)
They claim it also acts as an artificial electron donor to complex I-IV of the mitochondria
Oxygen-radical absorbance capacity assay for antioxidants
