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Description
Furthermore, cuprizone intoxication is a useful tool for studying the processes of remyelination, as withdrawal of cuprizone typically results in remyelination in the CNS of the intoxicated animal 21 , albeit at a significantly slower rate following chronic cuprizone intoxication 22
NCOA4 was downregulated in patients with ccRCC, and targeting NCOA4 to promote ferroptosis might be an effective therapeutic strategy[85]
A 31P NMR Study
It is not intended for human consumption, medical use, or diagnostic purposes
