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The R248W and R273H mutations attribute Mre11 nuclease-binding capacity to the p53 protein to prevent association of the Mre11-Rad50-NBS1 (MRN) complex with DNA double-strand breaks, thereby impairing ataxia-telangiectasia mutated (ATM) activation and the DNA damage response (Calheiros et al

Enzyme complexes of the respiratory chain of the oxidative phosphorylation, localized on the crests of the mitochondria, as the xanthines, the nicotinamide adenine dinucleotide phosphate (NADPH) oxidase and cytochrome P450, represent a source for a variety of ROS

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