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ROS-dependent activation of innate immune sensors, such as toll-like receptor 4 (TLR4) and NADPH oxidase (NOX2), initiates immune signaling through MAPKs, and PI3K/Akt pathways and induces the release of proinflammatory cytokines (IL-6, IL-8, IL-1, TNF-) [52] (Figure 2)

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This rapid central nervous system penetration is crucial for its potential to mitigate neuroinflammation and brain fog in Long COVID and ME/CFS patients

A mendelian randomization (MR) study explored the causal relationship between plasma Hcy levels and the risk of congestive heart failure and cardiomyopathy [42]