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Description
Jun 1996;16(4):257-63
Direct measurements of carnitine in skeletal muscles failed to show any elevation in the muscle carnitine concentration following 14 days of 4 g/day [21], or 6 g/day [22] LC ingestion
Addressing these dimensions will be essential for further understanding whether SLC25A45-dependent carnitine metabolism represents a universal adaptive mechanism or one selectively engaged under specific physiological or pathological conditions
Secondary deficiency can be characterized by substantial excretion of carnitine via the urine in acyl-carnitine form
