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In septic lung injury, elevated lactate levels exacerbate alveolar epithelial cell ferroptosis through lactylated histone H3 lysine 18 (H3K18la)-mediated upregulation of METTL3, which enhances m6A modification of ACSL4, ultimately contributing to the development of acute respiratory distress syndrome (82)

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193 In addition, Fusobacterium nucleatum can activate the RIPK1-mediated apoptosis pathway in UC, resulting in the destruction of tight junctions between cells, which in turn promotes the destruction of the intestinal barrier, inhibits the renewal of intestinal epithelial cells, and accelerates the progression of UC

Statistical analysis When conditions permitted, the study was divided into three arms based on the administered dose of GSH used to obtain the two-arm data (300 mg/d groups vs