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Notably, enhancing lipid utilization or myelin turnover in AD should be approached with caution, as chronic activation of lipid-buffer pathways may gradually compromise myelin integrity and promote demyelination, necessitating stage-specific strategies that balance short-term metabolic support with long-term sheath maintenance
In contrast, their expression was negatively correlated with that of TWIST1 , a mesenchymal marker, in these HCC patients (Fig
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However, further research is needed to fully understand DSIPs mechanisms and potential therapeutic applications
