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dosage of glutathione for copper deficiency improves testicular spermatogenesis through inhibiting oxidative stress, mitochondrial damage, and apoptosis induced by deposition in mice with Wilson disease A Targeted Metabolomic Assessment of

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Wu GH, Zhang YH, Yuan HH, Lan MB

dosage of glutathione for copper deficiency improves testicular spermatogenesis through inhibiting oxidative stress, mitochondrial damage, and apoptosis induced by deposition in mice with Wilson disease A Targeted Metabolomic Assessment of

Their biological function is determined by their bioavailability, their anti-inflammatory, and antioxidant properties, and other activities such as vasodilatory, anticancer effects, and anti-ischemia [58]

dosage of glutathione for copper deficiency improves testicular spermatogenesis through inhibiting oxidative stress, mitochondrial damage, and apoptosis induced by deposition in mice with Wilson disease A Targeted Metabolomic Assessment of

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dosage of glutathione for copper deficiency improves testicular spermatogenesis through inhibiting oxidative stress, mitochondrial damage, and apoptosis induced by deposition in mice with Wilson disease A Targeted Metabolomic Assessment of

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dosage of glutathione for copper deficiency improves testicular spermatogenesis through inhibiting oxidative stress, mitochondrial damage, and apoptosis induced by deposition in mice with Wilson disease A Targeted Metabolomic Assessment of

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