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[19] demonstrated that EGCG alleviated intestinal injury by regulating fatty acid metabolism, modulating the microbiota-tryptophan-aryl hydrocarbon receptor pathway (where gut microbes convert dietary tryptophan into indole derivatives that activate aryl hydrocarbon receptor (AhR, a ligand-activated transcription factor crucial for immune regulation and maintaining barrier function) to sustain mucosal IL-22 production and epithelial barrier integrity), inhibiting the Toll-like receptor-4 (TLR4)-nuclear factor kappa B (NF-B)/ mitogen-activated protein kinase (MAPKs)-NOD-like receptor family pyrin domain containing 3 (NLRP3) inflammatory cascade reaction and regulating nuclear factor erythroid 2-related factor 2 (Nrf2) signaling pathway

2a and Supplementary Table 2)

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