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These findings imply a significant positive effect with age and accelerate inflammation: following the step of initial dysbiosis and inflammation cascade, the altered GM and persistence of inflammation may act synergistically, prolonging the proinflammatory status
T cells require metabolic reprogramming during activation, resulting in various functional fates [46]
This review comprehensively examines the role and molecular mechanisms by which exercise alleviates atherosclerosis
The high-glycolysis phenotype of PC enhanced tumor progression by promoting epithelial mesenchymal transition, angiogenesis, and distant metastatic colonization (41)
