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It was observed the strain activated both B and T cells, and increased the level of anti-inflammatory cytokines such as IL-10 and reduced the level of pro-inflammatory cytokines (IL-1) (Hor et al., 2018)

Similarly, GHK-Cu showed notable findings in mouse models of ARDS, reducing TNF- and IL-6 expression, and demonstrating a protective effect against this rapid and potentially fatal inflammatory lung condition

At the molecular level, SRA directly bound Notch1 and suppressed its GS-ylation, thereby inhibiting the Notch pathway activation [112]
