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Singh, N

Dysbiosis may also promote excessive activation of T helper 17 (Th17) cells while impairing Treg function, thereby disrupting the balance between pro-inflammatory and anti-inflammatory immune responses (46, 47)

In summary, genetic risk factors for development of serious DILI at relatively low levels of APAP overdose seem likely to exist but the limited studies performed to date need to be followed up by genome-wide approaches using larger numbers of confirmed DILI cases or possibly further studies on volunteers showing transient ALT elevation

Availability of data and materials Data sharing is not applicable to this article as no datasets were generated or analysed during the current study